![]() ![]() Diabetes 40:405–412īenson J, Masor M (1994) Infant formula development: past, present and future. Mol Nutr Food Res 57:423–434īaynes JW (1991) Role of oxidative stress in development of complications in diabetes. Asian Pac J Trop Med 7:S348–S354īatista TM, Ribeiro RA, da Silva PM, Camargo RL, Lollo PC, Boschero AC, Carneiro EM (2013) Taurine supplementation improves liver glucose control in normal protein and malnourished mice fed a high-fat diet. Am J Physiol Endocrinol Metab 297:E620–E628īahmani M, Zargaran A, Rafieian-Kopaei M, Saki K (2014) Ethnobotanical study of medicinal plants used in the management of diabetes mellitus in the Urmia, Northwest Iran. Am J Physiol Heart Circ Physiol 301:H696–H703Īskwith T, Zeng W, Eggo MC, Stevens MJ (2009) Oxidative stress and dysregulation of the taurine transporter in high-glucose-exposed human Schwann cells: implications for pathogenesis of diabetic neuropathy. Int J Pharm Pharm Sci 4:134–147Īrrick DM, Sun H, Patel KP, Mayhan WG (2011) Chronic resveratrol treatment restores vascular responsiveness of cerebral arterioles in type 1 diabetic rats. ![]() Indian J Physiol Pharmacol 48:357–360Īnupama V, Narmadha R, Gopalakrishnan V, Devaki K (2012) Enzymatic alteration in the vital organs of streptozotocin diabetic rats treated with aqueous extract of Erythrina variegata bark. Cardiovasc Diabetol 10:61Īnthrayose C, Shashidhar S (2004) Studies on protein and taurine in normal, senile and diabetic cataractous human lenses. Comp Biochem Physiol C Toxicol Pharmacol 134:521–527Īnsar S, Koska J, Reaven PD (2011) Postprandial hyperlipidemia, endothelial dysfunction and cardiovascular risk: focus on incretins. Diabetes Res Clin Pract 67:3–21Īlvarado-Vásquez N, Zamudio P, Cerón E, Vanda B, Zenteno E, Carvajal-Sandoval G (2003) Effect of glycine in streptozotocin-induced diabetic rats. Food Chem Toxicol 71:116–121Īhmed N (2005) Advanced glycation end products-role in pathology of diabetic complications. J Perinat Med 30:281–286Īgca CA, Tuzcu M, Hayirli A, Sahin K (2014) Taurine ameliorates neuropathy via regulating NF-κB and Nrf2/HO-1 signaling cascades in diabetic rats. Life Sci 82:279–289Īerts L, van Assche FA (2002) Taurine and taurine-deficiency in the perinatal period. Taurine (2-aminoethane sulfonic acid) is a natural compound that has been studied in diabetes and diabetes-induced complications.Ībebe W (2008) Effects of taurine on the reactivity of aortas from diabetic rats. The worldwide threat of diabetes underscores the urgent need for novel therapeutic measures against this disorder. Several of the pathways are the advanced glycation end-product pathway, PI3-kinase/AKT pathway and mitochondrial apoptosis pathway. The mechanism of action of taurine cannot be explained by one pathway, as it has many effects. Taurine was shown to be effective against oxidative stress. Taurine has been observed to be effective in treatments against diabetic hepatotoxicity, vascular problems and heart injury in diabetes. Taurine treatment performed well against oxidative stress in brain, increased the secretion of required hormones and protected against neuropathy, retinopathy and nephropathy in diabetes compared with the control. Taurine improved glucagon activity, promoted glycemic stability, modified glucose levels, successfully addressed hyperglycemia via advanced glycation end-product control, improved insulin secretion and had a beneficial effect on insulin resistance. Diets in rats and humans have shown promising results. ![]()
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